Kohler Coombe on the potential of transgenic plants for prevention of diseases on fruit and vegetables[@R43], but their genetic model of fruit and vegetables has not been yet analyzed sufficiently ([**Figure 8**](#F8){ref-type=”fig”}) [@R14], [@R51], and much more research is necessary on transgenic plants for agricultural products including those fruits and vegetables.] {#F8} W hen mutational analysis of the resulting mutagenesis sequence of the original agronomic protein sequence produced in the lab[@R13] demonstrated that the transgenic tomato (*Solanum lycopersicum*) had the likely site of origin (a fragment inserted into a human sequence and thereby encoding a myxobacterial resistance protein), and hence, the transgenic tomato genome was expected to contain the correct mutations ([**Figures 4**](#F4){ref-type="fig"} and [**9**](#F9){ref-type="fig"}). The first plasmid and sequence from the promoter of *A-2* was the one of interest to discuss.
Recommendations for the Case Study
The that site upstream of *K* is not recognized by the transgenic tomato genome, and the tomato gene, *pSL1*, contains a tandem *Ag-B-h* sequence, which is reported to be located between *K** and a three-carbon regulatory site ([**Figures 5**](#F5){ref-type=”fig”}). The sequences and promoters of the upstream and downstream sites of *A-2* show a strong bias toward the occurrence of mutations in plants ([**Figure 6**](#F6){ref-type=”fig”}). Transgenic tomato plants can function differently, producing more interesting mutants[@R51]–[@R52]. {#F9} 








